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Objective : To study the possible relationship between mitochondrial DNA point mutations and hereditary ataxia.

目的: 探索线粒体DNA点突变与遗传性共济失调的关系.

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Conclusion: The point mutation in this area of mitochondrial DNA might not be related hereditary ataxia.

结论: 遗传性共济失调的发生、发展可能与该区域点突变无关.

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Objective : To construct enhanced cyan fluorescence protein ( ECFP ) lentiviral vector using the gene multipoint mutation.

目的: 通过多点突变构建增强型青色荧光蛋白 ( ECFP)慢病毒表达载体.

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Conclusion The model of Fgfr 3 Gly 374 Arg mutation causing achondroplasia in mice has been established successfully.

结论成功地建立了模拟人Fgfr3-Gly374Arg点突变所致软骨发育不全的小鼠模型.

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From our results , mitochondrial DNA mutation may be one of major factors aminoglycoside antibiotic induced deafness.

提示线粒体DNA点突变是导致该家系致聋的主要因素之一.

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