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Oligodendrocyte damage and subsequent axonal demyelination is a hallmark of this disease.

少突胶质细胞损害,继而轴突脱髓鞘为本病特征.

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Conclusion EAE is characterised by perivascular inflammatory cell infiltration and demyelination in white matter.

结论EAE的病理改变主要为血管周围炎性浸润及白质脱髓鞘.

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Significant endoneurial edema, inflammation reaction and demyelination can be seen in the DRGs and NRs.

自体髓核移植可致脊神经节、神经根明显的水肿 、 炎性反应、脱髓鞘等病理改变.

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